Jednym z największych skandali medycznych na przestrzeni ostatnich 100 lat - jest fakt, że od 1911 roku medyczny swiat wiedziedzial że stwardnienie rozsiane jest spowodowane przez bakterie .Big Pharma zakazala publikacji i badan na ten temat aby skorzystać z tak zwanych "pacjentów ze stwardnieniem rozsianym" ,
1913 Bullock W E (now Gye) MS agent in Rabbits Lancet 1185 1913
1917 Steiner Spirochetes The Cause of MS. Med Kiln
1918 Simmering Spirochetes in MS by Darkfield Micro
1918 Steiner G. Guinea Pig Inoculation with MS infectious agent from Human
1919 Steiner MS Agent Inoculation into Monkeys
1921 Gye F. MS Agent In Rabbits Brain 14:213
1922 Kaberlah MS Agent In Rabbits. Deutch Med Works
1922 Sicard MS Spirochetes in Animal Model. Rev Neurol
1922 Stepanopoulo Spirochetes in the CSF of MS Patients
1923 Schlossman MS Agent in Animal Model. Rev Neuro
1924 Blacklock MS Agent in Animals. Journal of Path and Bac
1927 Wilson The Rat as A Carrier of MS. British Med Journal
1927 Steiner G Understanding the Pathogenesis of MS
1928 Steiner Spirochetes in the Human Brain of MS Patients
1932 Rogers, Helen J. The question of silver cells as proof of the spirochetal theory of disseminated sclerosis. J. Neurol and Psychopathol. 13:50, 1932
1933 Simons Spirochetes in the CSF of MS Patients
1939 Hassin Spirochete-like formations in MS
1948 Adams Spirochetes within the Ventricle Fluid of Monkeys Inoculated from Human MS
1952 Steiner Acute Plaques in MS and The Pathogenic Role of Spirochetes as the Etiological Factor. Journal of Neuropathology Exp Med 11: No 4:343
1954 Steiner Morphology of Spirochaeta Myelophthora (Myelin Loving). MS Journal of Neuropathology and Exp Neurol 11:4 343
1954 Steiner G. Acute plaques in M.S., their pathogenetic significance and the role of spirochetes as the etiological factor. J. Neuropath. and Exp. Neur. 11:no 4:343, 1954
1957 Ichelson R. Cultivation of Spirochetes from Spinal Fluids of MS Cases with Negative Controls. Procl. Soc. Exp. Biol Med 70:411
1986 Gay D Dick G Is multiple sclerosis caused by an oral spirochaete? Lancet (1986 Jul 12) 2(8498):75-7
1988 Marshall V Multiple sclerosis is a chronic central nervous system infection by a spirochetal agent. Med Hypotheses (1988 Feb) 25(2):89-92
1986 (USA): Relapsing fever/Lyme disease – Multiple sclerosis. Medical Hypotheses, volume 21, issue 3, pages 335-343
2000 (Poland): Lyme borreliosis and Multiple sclerosis: Any Connection? A Seroepidemic study. Ann Agric Environ Med. issue 7, 141-143
2001 (Norway): Association between Multiple sclerosis and Cystic Structures in Cerebrospinal Fluid. Infect 29:315
2004 (Switzerland): Chronic Lyme borreliosis at the root of Multiple sclerosis – is a cure with antibiotics attainable?
2009 (Romania): Controversies in late Neuroborreliosis and Multiple sclerosis – case series
Multiple Sclerosis Societies.
But were those 15 researchers who said they found living Lyme bacteria (spirochetes) in the brains of a great majority of Multiple sclerosis patients all lying?
1911 Buzzard E F Spirochetes in M.S. Lancet 11:98 19111913 Bullock W E (now Gye) MS agent in Rabbits Lancet 1185 1913
1917 Steiner Spirochetes The Cause of MS. Med Kiln
1918 Simmering Spirochetes in MS by Darkfield Micro
1918 Steiner G. Guinea Pig Inoculation with MS infectious agent from Human
1919 Steiner MS Agent Inoculation into Monkeys
1921 Gye F. MS Agent In Rabbits Brain 14:213
1922 Kaberlah MS Agent In Rabbits. Deutch Med Works
1922 Sicard MS Spirochetes in Animal Model. Rev Neurol
1922 Stepanopoulo Spirochetes in the CSF of MS Patients
1923 Schlossman MS Agent in Animal Model. Rev Neuro
1924 Blacklock MS Agent in Animals. Journal of Path and Bac
1927 Wilson The Rat as A Carrier of MS. British Med Journal
1927 Steiner G Understanding the Pathogenesis of MS
1928 Steiner Spirochetes in the Human Brain of MS Patients
1932 Rogers, Helen J. The question of silver cells as proof of the spirochetal theory of disseminated sclerosis. J. Neurol and Psychopathol. 13:50, 1932
1933 Simons Spirochetes in the CSF of MS Patients
1939 Hassin Spirochete-like formations in MS
1948 Adams Spirochetes within the Ventricle Fluid of Monkeys Inoculated from Human MS
1952 Steiner Acute Plaques in MS and The Pathogenic Role of Spirochetes as the Etiological Factor. Journal of Neuropathology Exp Med 11: No 4:343
1954 Steiner Morphology of Spirochaeta Myelophthora (Myelin Loving). MS Journal of Neuropathology and Exp Neurol 11:4 343
1954 Steiner G. Acute plaques in M.S., their pathogenetic significance and the role of spirochetes as the etiological factor. J. Neuropath. and Exp. Neur. 11:no 4:343, 1954
1957 Ichelson R. Cultivation of Spirochetes from Spinal Fluids of MS Cases with Negative Controls. Procl. Soc. Exp. Biol Med 70:411
1986 Gay D Dick G Is multiple sclerosis caused by an oral spirochaete? Lancet (1986 Jul 12) 2(8498):75-7
1988 Marshall V Multiple sclerosis is a chronic central nervous system infection by a spirochetal agent. Med Hypotheses (1988 Feb) 25(2):89-92
1986 (USA): Relapsing fever/Lyme disease – Multiple sclerosis. Medical Hypotheses, volume 21, issue 3, pages 335-343
2000 (Poland): Lyme borreliosis and Multiple sclerosis: Any Connection? A Seroepidemic study. Ann Agric Environ Med. issue 7, 141-143
2001 (Norway): Association between Multiple sclerosis and Cystic Structures in Cerebrospinal Fluid. Infect 29:315
2004 (Switzerland): Chronic Lyme borreliosis at the root of Multiple sclerosis – is a cure with antibiotics attainable?
2009 (Romania): Controversies in late Neuroborreliosis and Multiple sclerosis – case series
Multiple Sclerosis Societies.
Every Western country has at least one MS Society. Each of those tax-exempt societies typically receives tens of millions of dollars in funding from various sources, year after year. The people running those societies usually award themselves CEO-level salaries and run them as one would run a highly commercial corporation. Advertising is used to solicit funds but if you don’t read ads then you’ll bump into them, one day, begging you for money on the street. For all those billions that have been pumped over the decades in those hundreds of MS societies worldwide, not a single one has ever done anything really useful for MS patients. The worst that could possibly happen for the bosses of those setups is that the cause of MS would become known. A known cause would either mean the development of either a cure or at least better symptom relievers, and that would rapidly result in the obsoleteness of their money making machine – the chicken that lays the golden eggs if you will. Such MS societies are working in concert with MS “researchers” employed by Big Pharma.
Big Pharma.
Multinational pharmaceutical corporations are the only ones doing MS research nowadays, mainly using donations to MS societies. Those multinationals decide which researchers get the cash. Researchers wanting to test the postulation of bacterial etiology of MS are shunned as if they were crackpots. Big Pharma makes billions a year on MS symptom relievers and they trickle millions down to their footsoldiers, the “MS experts”. A cure would be a severe financial blow. Even more so, because there is strong evidence that many other neurological illnesses are caused by germs as well. Because due to the phenomenon of immune privilege there is an inadequate immune response in the brain and spinal cord, making these organs the ideal place for certain slow-dividing spirochetal bacteria to entrench, multiply and cause lesions. The entire concept of antibiotic-resistant, hard-to-test-for chronic CNS infections leading to a plethora of neurological syndromes has to be suppressed and what can’t be suppressed will be craftily discredited. Better to give every expression of a neurological infection its own name such as “MS”, “Alzheimers”, “Parkinsons,” “ALS” and “Fibromyalgia”. And fund armies of ignorant “experts” to obfuscate the issue, whilst boycotting, firing, censoring, smearing and suing those few real experts that refuse to stay in line. Big Pharma is in business to make money, and money is made when people are ill, not when they’re healthy. Anyone standing in their way is relegated to the sidelines. Patents are being bought and shelved so that cures will never see the light of day.
Patient advocacy groups.
MS patient groups are, without exception, populated with clueless individuals for the simple reason that those who did their homework and read the relevant research have been ostracized by the group. They always were and they always will, because that’s how group dynamics works. As soon as you insist on voicing an opinion outside of the mainstream, no matter how well argued – you’ll be an outcast, a pariah. They don’t want rogue activists, “lone nutters”, giving them a bad name. Also the advocacy groups are raking in the dough and are run by folks whose main concern is that membership dues are paid in time. No MS, no advocacy group. Of course if there ever will emerge a lobby group insisting on more microbiological research pertaining Multiple sclerosis, they’ll be branded “lunatic fringe” and their efforts will be in vain.
MS “experts”.
Those “experts” get away with calling themselves thus, because Big Pharma gives them their seal of approval in the form of research grants and medical media exposure. However they are only experts in doing exactly what Big Pharma wants them to do: Obscuring the cause of Multiple Sclerosis! In return, the “experts” get regular cash injections for their “promising research” and other goodies such as all-in holidays to exotic destinations. There never will be a cure for MS until the scandal breaks and new antibiotics are developed that work better than the few currently available antibiotics that cross the blood-brain barrier. As it stands, it has been more than twenty years ago since any new antibiotic was developed. As soon as it was found that Minocycline helped with MS, its manufacturer, Lederle, tripled its price.
After long consideration I came to the conclusion that at least a crucial part of this debâcle was due to a real conspiracy – mainly a conspiracy of silence of those few MS researchers bright enough to realize that the cause for MS has been known for at least a hundred years. As is always the case with medical cover-ups, it continues to exist due to a mix of ignorance, indifference, cowardice and corruption. The saying goes: “Do not attribute to malice that what can be adequately blamed on ignorance”. All the “experts” really are interested in is being “experts”, not curing Multiple sclerosis. However it still is a conspiracy. It is completely normal for conspiracies to succeed because the lion share of the people who could point it out don’t care, are too lazy to get educated or feel too intimidated to stick out their necks. Microbiologist Tom Grier calls them cowards. The fact that most conspiracies are silently facilitated by an army of “useful idiots” with a stake in it being kept under the rug does not make it any less a conspiracy.
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Although significant information (reviewed in[43,44,70]) points to an infectious process in MS, this remains a controversial concept. As evidence emerges of new possible pathogens in MS, such as a new putative retrovirus,[71] these reports must be intensively examined and further studies initiated. Since most studies have found that the progressive form of MS, rather than relapsing-remitting forms of MS, were associated with chronic infections, infections might be more important in MS progression than in its inception. Various infections may also nonspecifically stimulate the immune system.[43] As in other neurodegenerative diseases, multiple factors appear to be involved in the pathogenesis of MS. Thus, like ALS, MS progression may turn out to be more likely linked to chronic infections, rather than its inception.Continue Reading
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Does the overall evidence suggest that chronic infections may be involved in the pathogenesis of neurodegenerative and neurobehavioral diseases? At the moment, the evidence is inconclusive. Some individuals can harbor chronic infections without any observable signs or symptoms, although the incidence of infection in such individuals is usually very low, only a few percent (for example[14-17]). Animal models have provided some additional insight,[117-119] but this area will require much more intensive investigation. Some information outside the area exists on the infection of nonhuman primates with neuropathologic microorganisms, such as Mycoplasma fermentans, which results in CNS infection and a fatal disease with neurological signs and symptoms.[166] Animal models that can be reproducibly infected with specific microorganisms to reproduce a similar disease will be an important resource. Future basic and clinical research may ultimately elucidate the involvement of chronic infections in the pathogenesis and progression of neurodegenerative and neurobehavioral diseases.
http://www.medscape.com/viewarticle/574944_9
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Abstract
A total of 769 adult neurological patients hospitalised in clinics and hospitals situated in the Lublin region (eastern Poland) were examined during the years 1997-2000 with ELISA test for the presence of anti-Borrelia burgdorferi sensu lato antibodies. A statististically significant (p=0.0422) relationship was found between the clinically confirmed diagnosis of multiple sclerosis and the positive serologic reaction with Borrelia antigen. Ten out 26 patients with multiple sclerosis (38.5%) showed positive serologic reaction to Borrelia, whereas among the total number of examined neurological patients the frequency of positive findings was twice as low (19.4%). The result suggests that multiple sclerosis may be often associated with Borrelia infection
http://www.ncbi.nlm.nih.gov/pubmed/11153045
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Lyme misdiagnosed as MS: Antibiotics cured “MS”:
---------------------------------------------------------------Although significant information (reviewed in[43,44,70]) points to an infectious process in MS, this remains a controversial concept. As evidence emerges of new possible pathogens in MS, such as a new putative retrovirus,[71] these reports must be intensively examined and further studies initiated. Since most studies have found that the progressive form of MS, rather than relapsing-remitting forms of MS, were associated with chronic infections, infections might be more important in MS progression than in its inception. Various infections may also nonspecifically stimulate the immune system.[43] As in other neurodegenerative diseases, multiple factors appear to be involved in the pathogenesis of MS. Thus, like ALS, MS progression may turn out to be more likely linked to chronic infections, rather than its inception.Continue Reading
-----------------------------------------
Does the overall evidence suggest that chronic infections may be involved in the pathogenesis of neurodegenerative and neurobehavioral diseases? At the moment, the evidence is inconclusive. Some individuals can harbor chronic infections without any observable signs or symptoms, although the incidence of infection in such individuals is usually very low, only a few percent (for example[14-17]). Animal models have provided some additional insight,[117-119] but this area will require much more intensive investigation. Some information outside the area exists on the infection of nonhuman primates with neuropathologic microorganisms, such as Mycoplasma fermentans, which results in CNS infection and a fatal disease with neurological signs and symptoms.[166] Animal models that can be reproducibly infected with specific microorganisms to reproduce a similar disease will be an important resource. Future basic and clinical research may ultimately elucidate the involvement of chronic infections in the pathogenesis and progression of neurodegenerative and neurobehavioral diseases.
http://www.medscape.com/viewarticle/574944_9
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Abstract
Interplay between susceptibility genes and environmental factors is considered important player in the genesis of multiple sclerosis (MS). Among environmental factors, a role for an infectious pathogen has long been considered central to the disease process. This opinion has support both from epidemiological data and the findings of immunological abnormalities in spinal fluid that reflect an immune response to an as yet undetermined antigen, possibly a pathogen, in the cerebrospinal fluid. Our review will outline the current understanding of the role of infection in the causation and progression of MS. We will review the data that point to an infectious cause of MS and consider the specific agents Chlamydophila (Chlamydia) pneumoniae, Human Herpes Virus 6, and Epstein-Barr Virus, that are implicated in either the development or progression of MS.
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Role of Chronic Bacterial and Viral Infections in Neurodegenerative, Neurobehavioral, Psychiatric, Autoimmune and Fatiguing Illnesses: Part 1
Abstract
Chronically ill patients with neurodegenerative, neurobehavioral and psychiatric diseases commonly have systemic and central nervous system bacterial and viral infections. In addition, other chronic illnesses where neurological manifestations are routinely found, such as fatiguing and autoimmune diseases, Lyme disease and Gulf War illnesses, also show systemic bacterial and viral infections that could be important in disease inception and progression or in increasing the number and severity of signs and symptoms. Evidence of Mycoplasma species, Chlamydia pneumoniae, Borrelia burgdorferi, human herpesvirus-1, -6 and -7 and other bacterial and viral infections revealed high infection rates in the above illnesses that were not found in controls. Although the specific roles of chronic infections in various diseases and their pathogeneses have not been carefully determined, the data suggest that chronic bacterial and/or viral infections are common features of progressive chronic diseases.
Abbreviations: Ab beta amyloid; AD Alzheimer’s disease; ADHD attention-deficit/hyperactivity disorder; ALS amyotrophic lateral sclerosis; ASD autism spectrum disorders; EBV Epstein-Barr virus; CFS chronic fatigue syndrome; CFS/ME chronic fatigue syndrome/myalgic encephalomyopathy; CI confidence interval; CMV cytomegalovirus; CSF cerebrospinal fluid; CNS central nervous system; ELISA enzyme linked immunoabsorbant assay; GWI Gulf War illnesses; HHV human herpes virus; HSV herpes simplex virus; PCR polymerase chain reaction; PD Parkinson’s disease
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Abstract
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