List of 700 Articles Citing Chronic Infection Associated with Tick-Borne Disease Compiled by Dr. Robert Bransfield
Dr. Robert Bransfield, past president of ILADS, has accumulated a list of 700 articles citing chronic infection associated with tick-borne diseases.
Patients exposed to tick-borne diseases sometimes have the progressive development of increasing symptoms, even after different antibiotic treatments. All of these symptoms cannot be explained by the presence of other unrelated conditions that are comorbid with and/or mimic tick-borne diseases. Although most agree that many of the progressive symptoms are immune mediated, there are two opposing views on the further cause of the disease process.
One hypothesis is infection is no longer present and there is speculation that some unknown self-perpetuating mechanism causes the intensification of chronic symptoms seen in these patients. The other hypothesis proposes that infection can persist which can cause intensifications of symptoms by provoking the immune system and possibly by other additional mechanisms.
The National Institute of Health recognizes many chronic diseases may eventually be proven to have an infectious basis and established the Human Microbiome Project which recognizes bacterial cells are ten times more prevalent than human cells in the human body.
With time there are increasing peer reviewed medical journal articles supporting the position that persistent tick-borne infection occurs and can contribute to causing the intensification of chronic symptoms which creates a serious burden of disease globally.
Below are some peer reviewed journal articles providing evidence supporting this scientific position.
The list contains over 700 articles and is divided into several different sections. The first section is a general list of articles supporting the evidence for persistent infection. The other sections support persistence as it pertains to psychiatric symptoms, dementia, autism, and congenital transmission. The list shall be periodically revised.
Download the article list [PDF]
http://www.ilads.org/ilads_news/2015/list-of-700-articles-citing-chronic-infection-associated-with-tick-borne-disease-compiled-by-dr-robert-bransfield/
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Professor Garth L. Nicholson is the President, Chief Scientific Officer and Research Professor at the Institute for Molecular Medicine in Huntington Beach, California. He received his B.S. in Chemistry from University of California in 1965 and his Ph.D. in Biochemistry and Cell Biology in 1970. He has published over 500 medical and scientific papers, edited 14 books and served on the Editorial Boards of 20 medical and scientific journals. He's a busy man, but found time to answer a few questions about Lyme disease.
Sue: Why is Lyme disease so difficult to diagnose and treat? The new Infectious Disease Society guidelines say, in essence, that it's a piece of cake.
Dr. Nicholson: Lyme Disease is not just about Borrelia burgdorferi. Lyme Disease is a collection of infections, often called Lyme co-infections. In the case of chronic Lyme disease, the most commonly found co-infections with Borrelia burgdorferi are bacteria: Mycoplasma species (found in 60-75% of Lyme cases), Ehrlichia species (found in 10-35% of cases), Bartonella species (found in 25-40%) and the protozoa Babesia (found in 10-20% of cases). There may also be other co-infections that we don't know about.
http://www.publichealthalert.org/interview-dr-garth-nicholson-phd-bacterial-co-infections-in-chronic-diseases.html
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Lyme Disease co-infections: Lottery anyone?
Time and time again, I hear people asking the question, "I've been treating my Lyme disease for years and I feel worse now than the day I started. Why?" Well, the answer might just be that treatment has been too focused on eradication of Borrelia, the causative agent of Lyme disease, and not focused enough on the numerous co-infections and other factors that are almost universally present.
http://www.publichealthalert.org/lyme-disease-co-infections-lottery-anyone.html
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Lyme disease can have devastating effects on the body and can trigger autoimmune diseases such as Hashimoto's Thyroiditis. The bacteria borrelia burgdorferi has been found to cross-react with thyroid tissue which triggers the autoimmunity by what is known as molecular mimicry. This basically means that your immune system tags an infectious agent but also attacks self-tissue that has a similar protein structure.
http://www.publichealthalert.org/the-lyme-thyroid-connection.html
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Dr. Robert Bransfield, past president of ILADS, has accumulated a list of 700 articles citing chronic infection associated with tick-borne diseases.
Patients exposed to tick-borne diseases sometimes have the progressive development of increasing symptoms, even after different antibiotic treatments. All of these symptoms cannot be explained by the presence of other unrelated conditions that are comorbid with and/or mimic tick-borne diseases. Although most agree that many of the progressive symptoms are immune mediated, there are two opposing views on the further cause of the disease process.
One hypothesis is infection is no longer present and there is speculation that some unknown self-perpetuating mechanism causes the intensification of chronic symptoms seen in these patients. The other hypothesis proposes that infection can persist which can cause intensifications of symptoms by provoking the immune system and possibly by other additional mechanisms.
The National Institute of Health recognizes many chronic diseases may eventually be proven to have an infectious basis and established the Human Microbiome Project which recognizes bacterial cells are ten times more prevalent than human cells in the human body.
With time there are increasing peer reviewed medical journal articles supporting the position that persistent tick-borne infection occurs and can contribute to causing the intensification of chronic symptoms which creates a serious burden of disease globally.
Below are some peer reviewed journal articles providing evidence supporting this scientific position.
The list contains over 700 articles and is divided into several different sections. The first section is a general list of articles supporting the evidence for persistent infection. The other sections support persistence as it pertains to psychiatric symptoms, dementia, autism, and congenital transmission. The list shall be periodically revised.
Download the article list [PDF]
http://www.ilads.org/ilads_news/2015/list-of-700-articles-citing-chronic-infection-associated-with-tick-borne-disease-compiled-by-dr-robert-bransfield/
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Sue: Why is Lyme disease so difficult to diagnose and treat? The new Infectious Disease Society guidelines say, in essence, that it's a piece of cake.
Dr. Nicholson: Lyme Disease is not just about Borrelia burgdorferi. Lyme Disease is a collection of infections, often called Lyme co-infections. In the case of chronic Lyme disease, the most commonly found co-infections with Borrelia burgdorferi are bacteria: Mycoplasma species (found in 60-75% of Lyme cases), Ehrlichia species (found in 10-35% of cases), Bartonella species (found in 25-40%) and the protozoa Babesia (found in 10-20% of cases). There may also be other co-infections that we don't know about.
http://www.publichealthalert.org/interview-dr-garth-nicholson-phd-bacterial-co-infections-in-chronic-diseases.html
--------------------------------------------------------------------------------------
Lyme Disease co-infections: Lottery anyone?
Time and time again, I hear people asking the question, "I've been treating my Lyme disease for years and I feel worse now than the day I started. Why?" Well, the answer might just be that treatment has been too focused on eradication of Borrelia, the causative agent of Lyme disease, and not focused enough on the numerous co-infections and other factors that are almost universally present.
http://www.publichealthalert.org/lyme-disease-co-infections-lottery-anyone.html
----------------------------------------------------------------------------------------
Lyme disease can have devastating effects on the body and can trigger autoimmune diseases such as Hashimoto's Thyroiditis. The bacteria borrelia burgdorferi has been found to cross-react with thyroid tissue which triggers the autoimmunity by what is known as molecular mimicry. This basically means that your immune system tags an infectious agent but also attacks self-tissue that has a similar protein structure.
http://www.publichealthalert.org/the-lyme-thyroid-connection.html
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